1
Why can't fatty acids directly increase blood glucose during fasting?
2
During beta‑oxidation of palmitic acid, how many molecules of NADH are produced in total (including the citric acid cycle)?
3
Which organ supplies the majority of glucose for the brain during prolonged fasting?
4
What is the primary energetic advantage of using fatty acids over glucose for heart metabolism?
5
Which step of gluconeogenesis directly bypasses the irreversible phosphofructokinase‑1 reaction of glycolysis?
6
During fasting, which amino acids are classified as glucogenic and thus can feed directly into the citric acid cycle?
7
What is the net ATP yield from complete oxidation of one palmitic acid molecule, after accounting for activation cost?
8
Which transport protein facilitates fatty acid entry into mitochondria for β‑oxidation?
9
Why does the brain increase its utilization of ketone bodies during prolonged fasting?
10
In the fed state, excess hepatic glucose is converted to which lipoprotein for export to peripheral tissues?
